I posted my comment at the end of the article and it got CENSORED. hilarious.
thanks for this critique of the vaccine. The first part dismissing the PCR testing is fine but as I pointed out the science is not limited to PCR. So why the author chooses to ignore the real science is beyond me - willful ignorance is quite common on the interwebs. haha. Then we run into this stat:
Vaccine - 8/18,198 (0.04% developed infection); Serious Side Effects: 0.6%
Placebo – 162/18,325 (0.88% developed infection); Serious Side Effects 0.5%
The author then states:
Therefore, the absolute risk reduction for Primary Efficacy Endpoint is 0.84%. (ie. 0.88-0.04)
the same Antibodies developed by the body to fight against the Covid spike protein (homologous with Syncythin 1 protein from human placenta) , could very well attack own cells for example placenta, causing either pregnancy or fetal problems or, infertility – when the autoimmunity prevents placenta from developing in the first place.
The retroviral SU/TM complex is half the size of that in Coronaviruses. Usually in evolution, smaller is later and more efficient. So the S1/S2 complex may be far more ancient than retroviruses, perhaps back beyond the Cretaceous/Tertiary (K/T) boundary at the great extinction event that occurred 65 million years ago. The viral attachment/fusion machine may have originated in some Jurassic Virological Park, and conserved in form and function ever since (Shi et al. 2018; Wertheim et al. 2013). The point being that its principal functional parts are extremely well preserved over time in each virus that uses the complex for attachment and entry. What one learns about one frequently applies to all of the others, albeit with some protein sequence variation.
SARS-CoV-2, the virus that causes COVID-19, is made up nearly 10,000 amino acids, of which around 1,300 are found in the spike protein. Syncytin-1 is made up of around 540 amino acids. Given that there are only 20 different types of amino acid, it isn’t surprising that many, many proteins share similarities.
To make a protein these long strings of amino acids are folded to form a 3D structure. For antibodies to mistakenly recognise syncytin-1 as SARS-CoV-2, there would have to be sufficient similarity of amino acids in these strings (which there isn’t) and the critical amino acids would need to be clustered together in the 3D molecule in a sufficiently similar and accessible way (which they aren’t).
The article inappropriately suggests (without adequate literature support) that there was a PCR-related two-month surge (both for April/May and Dec/Jan) in cancer deaths. This does not make any sense. The decline in May/June period was not related to any change in diagnostic testing criteria (i.e. PCR)…and cancer death rates do not follow these specific patterns (even though there is some seasonal variation for some cancers).
Influenza has been notably lower in the USA and other countries , so this is not likely a contributor to the pattern of COVID peaks that you suggest are caused by PCR excessive reports https://www.cdc.gov/flu/weekly/index.htm https://www.cdc.gov/mmwr/volumes/69/wr/mm6937a6.htm
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